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Carbonic anhydrase inhibitors primarily decrease the production of aqueous humor by inhibiting the enzyme carbonic anhydrase, which plays a crucial role in the bicarbonate ion formation necessary for the production of aqueous humor in the ciliary processes of the eye. When carbonic anhydrase is inhibited, there is reduced bicarbonate production, leading to less fluid being secreted into the aqueous humor. This results in a decrease in intraocular pressure, which is essential in the management of conditions like glaucoma.

The other options are less applicable in this context. While increasing production would be counterproductive in the clinical management of elevated intraocular pressure, claiming no effect does not align with our understanding of the physiological role of carbonic anhydrase in aqueous humor dynamics. Stabilizing production is also misleading since the inhibitors specifically work to reduce the overall production of humor rather than maintain a status quo.